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Hip Osteoarthritis

A chronic condition causing cartilage degeneration in the hip, resulting in pain, stiffness, and limited mobility. It occurs more frequently in older adults and individuals with predisposing factors.
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Definition – Epidemiology of Hip Osteoarthritis

The hip joint is one of the largest and most important joints in the human body, as it connects the trunk to the lower limb. It consists of the acetabulum (the articular surface of the pelvis), the femoral head, and the surrounding soft tissues (synovial membrane, joint capsule, ligaments). The articulating surfaces of the acetabulum and femoral head are covered with articular cartilage, a special, smooth, and resilient tissue that allows smooth movement with minimal friction.

Osteoarthritis is a chronic degenerative joint disease in which the articular cartilage gradually deteriorates. The loss of cartilage leads to abnormal movement between the femoral head and acetabulum, the formation of osteophytes (bony spurs in areas where bone does not normally exist), and, in advanced stages, direct bone-on-bone contact with destructive consequences for the articulating bones.

Hip osteoarthritis is estimated to affect about 10–20% of the population (approximately 90 per 100,000 per year). It usually appears after the age of 50, more frequently in women (especially in Southern Europe), and is less common in individuals of African descent.

Causes are varied and include age-related wear, previous injuries, congenital hip dysplasia, increased body weight, repetitive stress (due to occupation or sports), and hereditary predisposition.

Before

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After

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Symptoms and signs: Recognition and understanding of clinical manifestations.

Pain is the main symptom of the disease. It is typically located in the groin and may radiate to the ipsilateral knee or buttock. In the early stages, pain occurs after exertion, but later it may appear even at rest. Patients may have difficulty climbing stairs or walking, especially on uneven ground.

Stiffness or a sensation of “locking” of the joint appears in later stages. Patients find it difficult to bring the thigh towards the abdomen and have restricted internal and external hip rotation. Combined with pain, this limits daily activities, such as putting on socks and shoes or trimming toenails. Stiffness may be worse after rest and improve with a few steps.

Patients often present with limping (antalgic gait), either due to pain or because joint destruction has led to shortening of the affected limb and leg length discrepancy. The affected leg is often shorter, sometimes by more than two centimeters.

Many patients with hip osteoarthritis also suffer from low back pain, either due to leg length discrepancy or antalgic gait.

Diagnostic Procedures: Basic Examinations Used

A. Clinical examination

Diagnosis of hip osteoarthritis can be made by clinical assessment, taking into account the patient’s history (symptoms, age, occupation, activities, prior injuries, comorbidities, medications, family history) and clinical tests. Pain on hip rotation when the thigh is flexed towards the abdomen is the most common finding. Pain may also occur during hip adduction or abduction, with decreased range of motion, or with flexion contracture (persistent mild hip flexion preventing full extension).

Β. Imaging

A plain anteroposterior (AP) pelvic X-ray in standing position is the primary diagnostic tool. Main findings include:

  • Narrowing of the joint space: representing cartilage loss between the acetabulum and femoral head.
  • Subchondral sclerosis: areas of increased bone density (appearing whiter than surrounding bone).
  • Osteophytes: bony spurs at abnormal locations.
  • Bone cysts: cavities develop in the femoral head and/or the bones of the femoral head.
  • Deformation of the femoral head: in advanced stages, loss of sphericity, usually flattening.

In rare cases where plain radiographs do not confirm the diagnosis (e.g., early stages without significant structural damage), CT or MRI may be necessary.

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Therapeutic approaches: Management and treatment.

Α. Conservative treatment 

In the early stages of hip osteoarthritis, treatment is conservative and includes:

  • Simple analgesics: e.g. paracetamol - acetaminophen.
  • Non-steroidal anti-inflammatory drugs: e.g. naproxen, ibuprofen, coxibs.
  • Stronger analgesics: e.g. tramadol, codeine.
  • Walking aids: e.g., two crutches or one (on the opposite side of the affected hip), or a walker.
  • Physiotherapy: analgesic physiotherapy and kinesitherapy aimed at strengthening periarticular muscles and improving joint range of motion.
  • Weight loss: reduces the load on the affected joint
  • Activity modification: avoiding stairs and high-impact exercise such as running. Swimming and cycling are preferred.
  • Intra-articular corticosteroid injection: in selected cases, cortisone (alone or with local anesthetics) may provide short-term pain relief.
  • Injection of autologous biological agents: in some cases, intra-articular injection of PRP (Platelet-Rich Plasma) or stem cells harvested from the patient using specialized techniques may be beneficial.

Β. Surgical Treatment - ASI method

The definitive treatment for hip osteoarthritis is total hip arthroplasty, i.e., replacement of the joint with an artificial one.

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